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Ovarian cancer is the leading cause of death among gynecological malignancies. The complex multistep process for transformation from normal to cancerous cells includes a sequence of gene mutations and dysregulations. Tumor suppressor gene mutations with potential roles in the progression of ovarian cancer are generally linked to accumulation of mutations in the remaining functional allele. According to the «dominant-negative effect», these mutations can act by interfering with the functions of dominant active proteins, causing the dysregulation of the active protein. Alk5 and Tgfbr3, which play a role in TGF-β signaling, and Galectin-3, are of vital importance to ovarian cancer. Here we analyzed the expressions of these three genes in normal ovarian tissue and in 10 ovarian tumors
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